These results were in agreement TPCA-1 with those of Dogan et al. (2004) [25], who reported that the endometrial explants produced viable implants in 26 of 30 animals (86.6%), and that most of the explants were well vascularized. Analyses of the assessed microvessel density demonstrated that angiogenesis is higher in endometriotic lesions compared with the eutopic endometrium. Microvessel density was determined on the basis of vWF and α-SMA-positive vessels. The distribution of these vessel markers was more positive in stroma around the glands

in samples of endometriosis. Although no significant difference was observed between the vWF positive vessels in the two groups, the immunoreaction seemed to be more intense on day 15. It could be related to the microvessel size and that the endothelial Small molecule library chemical structure cell might not be adjacent to other pericyte

or vice versa. By other hand, the α-SMA-positive vessels were more numerous in samples of endometriosis at day 30 than at day 15. This difference is related to the fact that the most of the blood vessels are mature, as illustrated by their association with αSMA-positive pericytes [4]. These observations indicated that the development of new vessels is necessary for the establishment and the maintenance of the endometriotic lesions, and also that the selleck products neovessels formed were more mature in endometriosis after 30 days. Using the same markers in the nude-mouse model of endometriosis, Nap et al. (2004) [19] demonstrated that the development of new blood vessels remains of pivotal importance for the maintenance and growth

of endometriosis. One of the main characteristics of endometriosis is its inflammatory nature. It has been shown that cytokines released from immune cells play an important role in the pathogenesis of endometriosis, and many of these cytokines possess angiogenic activity [26, 27]. VEGF is the most-prominent and most-studied proangiogenic factor in endometriosis, and it is widely believed that VEGF is the main stimulus for angiogenesis and increased vessel permeability GNA12 in this disease [6]. Its activity depends on its binding to different receptors, such as VEGFR-2 (Flk-1). In our model, we were able to demonstrate that the expression of VEGF and Flk-1 is enhanced in endometriotic lesions as compared with controls. Their immunodistributions were observed focally in the cytoplasm of endothelial and glandular epithelial cells and diffusely in stromal cells, and were more intense in ectopic endometrial tissues. It was also observed that the number of activated macrophages (ED-1 positive cells) increased in endometriotic lesions. These results are in agreement with other studies that have shown that VEGF is strongly expressed by endometriotic lesions and activated macrophages [12, 28].

[35] India,

[35] India, Screening Library purchase Kashmir valley, all year round selleckchem Indian M, mean 29 years (n = 64) 38 ± 30, 41% < 25 Lower exposure to sunlight, female gender Indian F, mean 27 years (n = 28) 14 ± 11, 96% < 25 Gulvady et al. [44] India, Mumbai Indian M, 40–68 years, senior executives

(indoor workers; n = 86) 28% < 19 Earlier start of the workday Vupputuri et al. [43] India, Delhi (28° N) Asian Indian M, mean 43 years (for both men and women), urban, middle income, mostly working indoors (n = 51) 27 ± 17 – Asian Indian F, mean 43 years (for both men and women), urban, middle income, mostly housewives (n = 54) 22 ± 12 Harinarayan [65] India, Tirupati (13° N), all year round Indian F, mean 54 years, postmenopausal (n = 164) 37 ± 18, 30% < 25 Higher CHIR98014 mouse dietary calcium intake, higher dietary phytate intake, higher phytate to calcium ratio Harinarayan et al. [21] India, around Tirupati (13° N), winter to summer (Jan–Jul) Indian, mean 44 years, rural (n = 191) 53 ± 06, 03% < 25 Urban subject, lower dietary calcium intake, higher phytate to calcium ratio Indian, mean 46 years, urban (n = 125) 34 ± 07, 35% < 25 Goswami et al. [18] India, Dehli (28° N), in winter or

summer Indian M, mean 25 years, soldiers, winter oxyclozanide (n = 31) 47 ± 12 Less exposure to sunlight, more skin pigmentation, winter season Indian M (58%)+F, mean 23 years, physicians and nurses, winter (n = 19) 08 ± 03 Indian M (67%)+F, mean 43 years, depigmented persons, winter (n = 15) 18 ± 11 Indian M (58%)+F, mean 24 years, physicians and nurses, summer (n = 19) 18 ± 08 Pregnant women Sahu et al. [36] India, Barabanki

district, 32 km from Lucknow (27°), all year round Indian, rural, mean 27 years (n = 139) 38 ± 20, 32% < 25 Lower summer sun exposure, measurement in winter Farrant et al. [66] India, Mysore (South India) at the 30th week of pregnancy Indian, mean 24 years (n = 559) Median 38, 31% < 28 nmol/l Taking calcium and vitamin D at recruitment, measurement in Mar–Aug Bhalala et al. [45] Western India, at the 37th week of pregnancy, all year round Indian, 20–35 years, middle income group (n = 42) 57 ± 27 Lower serum 25(OH)D in mother → lower serum 25(OH)D in cord blood Cord blood (n = 42) 48 ± 24 Sachan et al. [46] India, Lucknow (27° N), before labor, autumn Indian, total group (n = 207) 43% < 25 – Indian, urban (n = 140) 35 ± 24 Indian, rural (n = 67) 35 ± 22 Goswami et al. [18] India, Dehli (28° N), in summer Indian, mean 23 years, poor socioeconomic class (n = 29) 22 ± 11 – Children Sahu et al.

Static magnetic properties of the top films of the

Static magnetic properties of the top films of the nanobrush are shown in Figure  4. The (100)-textured sample shows the smallest coercivity and a good aspect ratio. For the FeNi film deposited on AAO templates, surface defects may destroy the soft magnetic properties. The magnetic moment distribution induced by the interface coupling effect conveys different characteristics, which may result in different performances of magnetoimpedance effect Cilengitide datasheet of the nanobrush. The insets of Figure  4 show the distribution of magnetic moments of the

top film in the nanobrush. The nanobrush combined with permalloy film and hcp Co nanowires is used during simulation. The thickness of the permalloy film and the diameter of Co nanowires are both 50 nm. An external field applied in the plane of the film is 50 Oe. The direction

of magnetic moments is denoted by the arrows. As shown in the inset, the magnetic moments of a KPT-8602 datasheet single film lie in the plane. When an external field was applied, the magnetic moments turn to the field direction. Transverse moments selleck chemicals can hardly be found. However, for the films of the nanobrush, a strong exchange coupling effect takes place at the interface of the nanofilm and nanowire array, leading to a vortex distribution of magnetic moment, and lot moments turn to be perpendicular to the applied field. Thus, the MI effect may be intensified due to the transverse component magnetic moments. For the (100) texture, magnetic moments distribute perpendicular to the long axis of nanowires. At the interface, planar vortex distribution of film moments is induced by the exchange coupling effect. Most transverse Tryptophan synthase magnetic moments will enhance the transverse permeability when an external field is applied. By contrast, the magnetic moments in (002) texture nanowires are along the long axis, and the induced vortex distributions

will be perpendicular to the film plane. Although many transverse moments have been observed, the perpendicular moments may block the increase of transverse moments and reduce the transverse permeability. Figure 4 Static magnetic properties of nanobrushes with different textures. Micromagnetic simulations of the top surface magnetic properties of the nanobrush are shown in the inset. Figure  5 shows the MI ratio under different applied fields of the nanobrush in combination with the FeNi film and 20-nm (100)-textured cobalt nanowires at different frequencies (f = 10, 30, 70, and 100 MHz). As the inset shows, the applied field is along the direction of the ac current, which is parallel to the FeNi film. On the one hand, with the externally applied magnetic field increasing, the MI ratio increases sharply and an obvious change of the MI ratio takes place in small fields. The MI curves can be explained by the magnetization rotation model [29], in which the transverse magnetic permeability plays an important role.

Data analysis was performed using manufacturer’s program and is b

Data analysis was performed using manufacturer’s program and is based on the ddCt method, with normalization of the raw data to the panel of housekeeping genes provided in the array. The genes showing modulation Stem Cells inhibitor by 1.5 fold up or down were only selected for further analysis. Functional annotations of the selected genes were carried out by the

bioinformatics software David for Bioinformatics. Three independent experiments with a pool of 2 donors each were analyzed. Statistical analysis Statistical evaluation of the data was done using GraphPad Prism 5 software. Student t-test was performed for simple comparison between 2 means. For multiple comparisons, the results were analysed by two-way ANOVA followed by Bonferoni’s post-test. p < 0.05 was considered statistically significant. All shown data are representative for at

least 3 independent experiments. Results Chlamydia trachomatis infect monocytes and monocyte-derived DCs in a see more comparable manner Monocytes isolated from human peripheral blood mononuclear cells (PBMCs) and monocyte-derived DCs were infected with C. trachomatis serovars Ba, D and L2 (Figure 1). Results show that all the three serovars were capable of infecting both the monocytes and DCs and form GM6001 chemical structure inclusions as detected by immunofluorescence microscopy 2 days post infection (p.i.). However, the inclusions were smaller in size compared to typical inclusions that have been reported in

HeLa cells (Additional file 2: Figure S2). The inclusion morphology and staining intensity varied between the infected monocytes and DCs. Figure 1 Immunofluorescence microscopy of infected monocytes and monocyte-derived Dendritic cells (DCs). Monocytes (upper panel) and monocyte-derived DCs (lower panel) were infected with C. trachomatis serovars Ba, D and Adenosine triphosphate L2 (MOI-3) for 2 days. Chlamydial inclusions (green) were stained with FITC conjugated anti-chlamydia LPS antibody and counterstained with Evans Blue. Pictures were taken at 63X magnification with Leica DMLB. The figures are representative of 3 independent experiments. In monocytes, the percentage of infected cells were comparable among the three serovars and did not seem to change even when the infection duration was extended to 3 days (Table 1). For DCs, the percentage of infected cells were similar for serovars Ba and D but serovar L2 showed a higher infection rate as compared to the other two (Table 1). However, the infection rate declined remarkably for all the three serovars when infected for 3 days. The infection rate was nevertheless much lower in both monocytes and DCs than in HeLa. Mock controls were prepared for each round of experiments which showed absence of chlamydial antigens in the donors (Additional file 3: Figure S3). Table 1 Comparison of infection rate in monocytes and monocyte-derived DCs infected with C.

Shigeta M, Tanaka G, Komatsuzawa H, Sugai M, Suginaka H, Usui T:

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10 S

Toxicology in Vitro 2001, 15:591–595.PubMedCrossRef

10. Spain DA, Miller FB: Education and training of the future surgeon in acute care surgery: trauma, critical care, and emergency surgery. Am J Surg 2005, 190:212–217.PubMedCrossRef 11. Gilbody J, Prasthofer AW, Ho K, Costa ML: The use and effectiveness of cadaveric workshops in higher surgical training: a systematic review. Ann R Coll Surg Engl 2011, 93:347–352.PubMedCrossRef 12. Cherry RA, Ali J: Current Concepts in Simulation-Based Trauma Education. Nirogacestat order J Trauma 2008, 65:1186–1193.PubMedCrossRef 13. Anastakis DJ, Regehr G, Reznick RK, Cusimano M, Murnaghan J, Brown M, Hutchison C: Assessment of Technical Skills Transfer from the Bench Training Model to the Human Model. Am J Surg 1999, 177:167–170.PubMedCrossRef 14. Donias HW, Schwartz T, Tang DG, DeAnda A Jr, Tabaie HA, Boyd DW, Karamanoukian HL: A porcine beating heart model for robotic coronary artery surgery. Heart Surg Forum 2003, 6:249–253.PubMed 15. Hishikawa S, Kawano M, Tanaka

H, Konno K, Yasuda Y, Kawano R, Kobayashi E, Lefor AT: Simulation improves operator confidence but not performance of tube thoracostomy by medical students in a porcine model: A prospective controlled trial. Am Surg 2010, 76:73–78.PubMed Competing interests The authors declare that they have no competing interests. Authors’ contributions YI: Conceived the trial, conducted the training, collected

buy Stattic and analyzed data, prepared the manuscript, SH: Conducted the training, collected the data, prepared the manuscript, TM: conducted the training, collected and analyzed the data, KY: conducted the training, collected and analyzed the data, MS: conceived the trial, analyzed the data, prepared the manuscript, AL: conceived the trial, Analyzed the data, preparation of manuscript, All authors read and approved the final manuscript.”
Vactosertib nmr Background Typhoid fever, a severe febrile illness caused primarily by a gram negative bacillus Salmonella typhi, has continued to be a public health problem in many developing countries [1, 2]. Typhoid infection is generally transmitted by faeco-oral route and may occasionally lead to an epidemic, particularly in areas with poor sanitation and limited availability of clean, Y-27632 price potable water [1–4]. It is a global health problem that can have a devastating impact on resource-poor countries like Tanzania and it is estimated that more than 33 million cases of typhoid fever occur annually causing more than 500,000 deaths [2, 5, 6]. While control of the infection has been achieved in developed countries by effective public health measures, developing countries continue to bear the burden of the disease, principally because many communities still fall short of standards for drinking water, hygiene and sanitation [2, 7, 8].

Management of hyperglycemia in type 2 diabetes: a patient-centere

Management of hyperglycemia in type 2 diabetes: a patient-centered approach. Position statement of the American Diabetes

Association (ADA) and the European Association for the Study of Diabetes (EASD). Diabetes Care. 2012;35:1364–79. doi:10.​2337/​dc12-0413dc12-0413.PubMedCentralPubMedCrossRef 20. Malerczyk V, Badian M, Korn A, Lehr KH, Waldhausl W. Dose linearity assessment of glimepiride (Amaryl) tablets in healthy volunteers. Drug Metabol Drug Interact. 1994;11:341–57.PubMedCrossRef 21. Rosenkranz B. Pharmacokinetic BYL719 clinical trial basis for the safety of glimepiride in risk groups of NIDDM patients. Horm Metab Res. 1996;28:434–9. doi:10.​1055/​s-2007-979833.PubMedCrossRef 22. Sanofi-aventis (2013). AMARYL (glimepiride) tablets. FDA. http://​www.​accessdata.​fda.​gov/​drugsatfda_​docs/​label/​2013/​020496s027lbl.​pdf. Accessed 3 Dec 2013. 23. Declaration of Helsinki. Ethical principles for medical research involving human subjects. World Medical Association; 2008. http://​www.​wma.​net/​en/​30publications/​10policies/​b3/​17c.​pdf.

MM-102 in vivo Accessed 3 Dec 2013. 24. ICH. Guideline for Good Clinical find more Practice E6 (R1). ICH Harmonised Tripartite Guideline; 1996. http://​www.​ich.​org/​fileadmin/​Public_​Web_​Site/​ICH_​Products/​Guidelines/​Efficacy/​E6_​R1/​Step4/​E6_​R1_​_​Guideline.​pdf. Accessed 2 Dec 2013. 25. Rowland M, Tozer T. Clinical pharmacokinetics and pharmacodynamics: concepts and applications. Philadelphia: Lippincott Williams & Wilkins; 2011. 26. FDA. Guidance for Industry. Drug interaction studies: study design, data analysis, implications for dosing, and labeling recommendations. US Department of Health and Human Services, Food and Drug Administration, Center for Drug Evaluation and Research (CDER); 2012. http://​www.​fda.​gov/​downloads/​Drugs/​GuidanceComplian​ceRegulatoryInfo​rmation/​Guidances/​UCM292362.​pdf. Accessed 3 Dec 2013. 27. Chen

L, Magliano DJ, Zimmet PZ. The worldwide epidemiology of type 2 diabetes mellitus–present and future perspectives. Nat Rev Endocrinol. selleck chemicals 2012;8:228–36. doi:10.​1038/​nrendo.​2011.​183nrendo.​2011.​183.CrossRef 28. CDC. National diabetes fact sheet. US Department of Health and Human Services; 2011. http://​www.​cdc.​gov/​diabetes/​pubs/​pdf/​ndfs_​2011.​pdf. 29. Stratton IM, Adler AI, Neil HA, Matthews DR, Manley SE, Cull CA, Hadden D, Turner RC, Holman RR. Association of glycaemia with macrovascular and microvascular complications of type 2 diabetes (UKPDS 35): prospective observational study. BMJ. 2000;321:405–12.PubMedCentralPubMedCrossRef 30. Nathan DM, Buse JB, Davidson MB, Ferrannini E, Holman RR, Sherwin R, Zinman B. Medical management of hyperglycemia in type 2 diabetes: a consensus algorithm for the initiation and adjustment of therapy: a consensus statement of the American Diabetes Association and the European Association for the Study of Diabetes. Diabetes Care. 2009;32:193–203. doi:10.​2337/​dc08-9025.

Blankenship (USA), Ralph Bock (Germany), Julian Eaton-Rye (New Ze

Blankenship (USA), Ralph Bock (STA-9090 Germany), Julian Eaton-Rye (New Zealand), Wayne Frasch (USA), Johannes Messinger (Sweden), Masahiro Sugiura (Japan), Davide Zannni (Italy), and Lixin Zhang (China). In view of inclusion of “Bioenergy and Related Processes” to the title of our Series, we seek suggestions of names of scientists who may be suitable for the future Board of Consulting Editors. Govindjee and I thank all who have served as editors or authors and hope that photosynthesis research will benefit for many years because of the community

effort to document A dvances in P hotosynthesis and R espiration Including Bioenergy and Related Processes.”
“Introduction AZD1480 mouse Natural photosynthesis achieves the conversion of solar energy with a remarkably small set of cofactors. Photosynthetic proteins use (bacterio)chlorophylls (BChls) and carotenoids (Car) both for light-harvesting and charge separation,

implying that the functional programming of the pigment chromophores is encoded in their conformation, local environment, and dynamics and is not due to their chemical structure per se. While the architecture of the photosynthetic reaction centers that leads to directional electron transfer is common to all photosynthetic organisms, there is much to be learned about the structure–function relations from the variability in photosynthetic antenna systems, as evolution has led to fundamentally different architectures for harvesting the light, depending on the variability of environmental sun light conditions. One intriguing puzzle that is currently S63845 solubility dmso attracting widespread attention is the molecular basis underlying the photophysical mechanism of nonphotochemical quenching (NPQ), a photoprotective switching mechanism that Montelukast Sodium protects oxygenic species at high sun light conditions while optimally photosynthesizing at

low light intensities. During the past three decades, many structures of photosynthetic membrane proteins have been resolved at high resolution by crystallography, but the details of the structure–function interactions and how cofactors are programmed for their function remain to be elucidated. Solid-state NMR may not outperform crystallography for resolving membrane protein structures, but the technique has compelling advantages when it comes to resolving atomic details of pigment–protein interactions in a flexible protein environment. Better understanding of the structure–function motifs across antenna complexes and photosynthetic species in an evolutionary context will provide knowledge on common denominators of functional mechanisms in natural photosynthetic systems. This will guide the design of novel artificial constructs in which dye molecules are preprogrammed in the ground state by engineering of their scaffolding environment to perform the different tasks of light harvesting, charge separation, and photoprotection (de Groot 2012).

In response to a plant signal present in nodules, three receptor-

In response to a plant SN-38 signal present in nodules, three receptor-like adenylate cyclases CyaD1, CyaD2 and CyaK synthesize the secondary messenger molecule 3′, 5′cAMP. 3′, 5′cAMP together with the Crp-like transcriptional activator Clr in turn promote transcription of the target gene smc02178, of unknown biochemical function [3]. We have recently found that this cascade contributes to the autoregulation of the symbiotic interaction. Specifically, activation of the cAMP cascade in nodules inhibits, by a mechanism that remains to be elucidated, secondary infection by rhizospheric bacteria.

This control is lost in either a triple cyaD1cyaD2cyaK mutant, a clr or a smc02178 mutant resulting in a hyper-infection phenotype on plants–ie an abundance of EPZ015938 cell line abortive ITs on roots–as a consequence of a relaxed control of secondary infection [3]. The concentration of the second messenger 3′, 5′cAMP in cells is controlled at the level of its synthesis by ACs and/or by its degradation Lazertinib cost to 5′AMP by phosphodiesterases (PDEs). PDEs are a superfamily of enzymes divided in three, non-homologous, main classes. All mammalian PDEs as well as several enzymes identified in Drosophila, Caenorhabditis and Saccharomyces cerevisiae belong to class I, whose conserved

carboxy-terminal catalytic domain contains two invariant motifs H(X)3H(X)25-35D/E [17]. Class II PDEs are enzymes from Saccharomyces cerevisiae, Dictyostelium discoideum, Schizosaccharomyces pombe, C. albicans, and Vibrio fischeri[17]. This class of enzymes shares the conserved motif HXHLDH. Class III PDEs belong to the superfamily of metallophosphoesterases [18]. They share the conserved sequence motif D-(X)n-GD(X)n-GNH[E/D]-(X)n-H-(X)n-GHXH

as well as a βαβαβ secondary structure signature Benzatropine [17]. Here we report on the characterization of a class III PDE from S. meliloti (SpdA, SMc02179) that we anticipated from the localization of the spdA gene at the cyaD1 locus to be involved in signal termination by turning-over the secondary messenger 3′, 5′cAMP. We have found that purified SpdA had actually no detectable activity against 3′, 5′cAMP and, instead, had high activity on the structural isomer 2′, 3′cAMP, which may occur in cells as a by-product of RNA degradation [19]. We demonstrated that, contrary to 3′, 5′cAMP that promoted Clr binding to a cognate binding-site, 2′, 3′cAMP bound unproductively to Clr. Although SpdA biological function remains to be established, we present circumstantial evidence that SpdA may insulate 3′, 5′cAMP-mediated signaling from 2′, 3′-structural isomers. Results SpdA, a putative PDE Inspection of the cyaD1 locus (Figure 1A), that contains the clr gene as well as the clr–target gene smc02178, pointed to the smc02179 gene product as a potential PDE that we subsequently coined SpdA.

Bacterial cells were lysed using 2 μL lysostaphin (1 mg/mL, Sigma

Bacterial cells were lysed using 2 μL lysostaphin (1 mg/mL, Sigma) in a 250 μL bacterial suspension and DNA was digested with SmaI (TaKaRa). Pulsed-field gel electrophoresis (PFGE) was performed using the CHEF-DR III system (Bio-Rad) on a 1% agarose (Cambraex Bio Science, Rockland) in 0.5 X TBE buffer (45 mM Tris-borate, 1 mM EDTA) for a run time of 18 h, with a voltage of 6 V/cm, pulses ramped from 4.0 to 40.0 s, at an angle of 120°. The standard strain H9812 (XbaI enzyme) was used as the electrophoresis marker. Gels were stained with 1 μg/mL ethidium bromide Erismodegib cell line for 30 min, washed in water for 30 min, and photographed using a Gel Doc 2000 (Bio-Rad). Band patterns were analyzed with BioNumerics version

3.0 (Applied Maths BVBA, Belgium) with the Dice coefficient and UPGMA clustering at 1.5% band tolerance. Acknowledgments We thank Research Fellow Wei Li and Associate Research Fellow Jinhua Cui of PulseNET China of Institute for Infectious Disease Control and Prevention (ICDC) of Chinese Center for Disease Control and Prevention (China CDC) for helping in PFGE techniques in the epidemiological study. References 1. Freney J, Brun Y, Bes M, Meugnier H, Grimont F, Grimont PAD, Nervi C, Fleurette J: Staphylococcus lugdunensis sp. nov. and Staphylococcus schleiferi sp. nov., Two Species from Human Clinical Specimens. Int J Syst

Bacteriol 1988, 38:168–172.CrossRef 2. Bieber L, Kahlmeter G: Staphylococcus lugdunensis in several niches of the normal skin flora. Clin Microbiol Infect 2010, during 16:385–388.PubMedCrossRef 3. Anguera click here I, Del Río A, Miró JM, et al.: Staphylococcus lugdunensis infective endocarditis: description of 10 cases and analysis of native valve, prosthetic valve, and pacemaker lead endocarditis clinical profiles. Heart 2005, 91:e10.PubMedCrossRef 4. Grupper M, Potasman I, Rosner I, Slobodin G, Rozenbaum M: Septic arthritis due to Staphylococcus lugdunensis

in a native joint. Rheumatol Int 2010, 30:1231–1233.PubMedCrossRef 5. Mei-Dan O, Mann G, Steinbacher G, Ballester S, Cugat R, Alvarez P: Septic arthritis with Staphylococcus lugdunensis following arthroscopic ACL revision with BPTB allograft. Knee Surg Sport Traumatol Arthrosc 2008, 16:15–18.CrossRef 6. Pada S, Lye DC, Leo YS, Barkham T: Utility of 16 S ribosomal DNA sequencing in the diagnosis of Staphylococcus lugdunensis native valve infective endocarditis: case report and literature review. IJID Off Publ Int Soc Infect Dis 2009, 13:e511-e513. 7. Kleiner E, Monk AB, Archer GL, Forbes BA: Clinical significance of Staphylococcus lugdunensis isolated from routine cultures. Clin Infect Dis 2010, 51:801–803.PubMedCrossRef 8. Tee WSN, Soh SY, Lin R, Loo LH: Staphylococcus lugdunensis Carrying the mecA Gene Causes Catheter-Associated ATM inhibitor Bloodstream Infection in Premature Neonate. J Clin Microbiol 2003, 41:519–520.PubMedCrossRef 9.